New Scientific Findings On Calcium Channel Blockers Raise Health Concerns

Long-term vitamin C deficiency is accompanied by an insufficient production of collagen. This is especially harmful for the artery walls as damage to their structure leads to the build-up of atherosclerotic deposits.

Researchers at the Dr. Rath Research Institute have shown that, in addition to blocking the cellular entry of calcium and other ions (sodium, potassium), channel blockers also obstruct the uptake of vitamin C by the cells. This can generate serious health problems, as vitamin C is an essential nutrient for the production of collagen, the key stability molecule in the body for the blood vessel walls and connective tissue.

Heart patients are given calcium channel blockers for long periods of time, if not for the rest of their lives. The consequence is longterm vitamin C deficiency, which can cause serious damage to the cells of the artery walls. This is followed by the build-up of atherosclerotic deposits, the cause of heart attacks and strokes.

Chronic vitamin C deficiency is linked to an insufficient production of collagen. Collagen is especially important for stable blood vessels. As a consequence of this collagen deficiency, millions of small cracks and crevices develop along the artery walls.

As a result, fat molecules and proteins are deposited inside the artery walls. Their deposition is initially a very useful repair mechanism that prevents blood loss through the structurally impaired walls.

However, if vitamin C deficiency becomes chronic, this repair process continues over an extended period of time, resulting in the formation of arteriosclerotic plaques to counterbalance the impairment of the artery walls. Ultimately, arteriosclerotic plaques occurring in coronary arteries lead to heart attack, or to stroke if they appear in the cerebral arteries.

Calcium antagonists never block cell wall channels completely, thus allowing a small portion of vitamin C to enter the cells. However, this portion is not enough to ensure the health and stability of the artery walls.

The study shows that collagen production is inhibited by calcium channel blockers. Nifedipine has the most pronounced inhibitory effect on collagen production by the vascular wall cells. Nifedipine is the best known representative of the calcium channel blockers drug group.

Calcium channel blockers inhibit collagen production

The study shows that collagen production is inhibited by calcium channel blockers. Nifedipine has the most pronounced inhibitory effect on collagen production by the vascular wall cells. Nifedipine is the best known representative of the calcium channel blockers drug group.